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Hippocampal alterations in glutamatergic signaling during amyloid progression in AβPP/PS1 mice
Our previous research demonstrated that soluble amyloid-β (Aβ)(42), elicits presynaptic glutamate release. We hypothesized that accumulation and deposition of Aβ altered glutamatergic neurotransmission in a temporally and spatially dependent manner. To test this hypothesis, a glutamate selective mic...
Uloženo v:
| Vydáno v: | Sci Rep |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Nature Publishing Group UK
2020
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7467928/ https://ncbi.nlm.nih.gov/pubmed/32879385 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-71587-6 |
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