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A cell permeant phosphopeptide mimetic of Niban inhibits p38 MAPK and restores endothelial function after injury
Vascular injury leads to membrane disruption, ATP release, and endothelial dysfunction. Increases in the phosphorylation of p38 mitogen‐activated protein kinase (p38 MAPK) and decreases in the phosphorylation of Niban, a protein implicated in ER stress and apoptosis, are associated with vascular inj...
Kaydedildi:
| Yayımlandı: | FASEB J |
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| Asıl Yazarlar: | , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
John Wiley and Sons Inc.
2020
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7383822/ https://ncbi.nlm.nih.gov/pubmed/32396246 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201902745R |
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