A cell permeant phosphopeptide mimetic of Niban inhibits p38 MAPK and restores endothelial function after injury

Vascular injury leads to membrane disruption, ATP release, and endothelial dysfunction. Increases in the phosphorylation of p38 mitogen‐activated protein kinase (p38 MAPK) and decreases in the phosphorylation of Niban, a protein implicated in ER stress and apoptosis, are associated with vascular inj...

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Detalles Bibliográficos
Publicado en:FASEB J
Autores principales: Yim, Tsz Wing, Perling, Daniel, Polcz, Monica, Komalavilas, Padmini, Brophy, Colleen, Cheung‐Flynn, Joyce
Formato: Artigo
Lenguaje:Inglês
Publicado: John Wiley and Sons Inc. 2020
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Research Articles
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC7383822/
https://ncbi.nlm.nih.gov/pubmed/32396246
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201902745R
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