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IL-23 promotes a coordinated B-cell germinal center program for class-switch recombination to IgG2b in BXD2 mice
IL-23 promotes autoimmune disease including Th17 CD4 T-cell development and autoantibody (autoAb) production. Here, we show that a deficiency of the p19 component of IL-23 in the autoimmune BXD2 (BXD2-p19(−/−)) mouse leads to a shift of the follicular T-helper cell program from Tfh-IL-17 to Tfh-IFN-...
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| Publicat a: | J Immunol |
|---|---|
| Autors principals: | , , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7374065/ https://ncbi.nlm.nih.gov/pubmed/32554431 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.2000280 |
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