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Stress-mediated exit to quiescence restricted by increasing persistence in CDK4/6 activation
Mammalian cells typically start the cell-cycle entry program by activating cyclin-dependent protein kinase 4/6 (CDK4/6). CDK4/6 activity is clinically relevant as mutations, deletions, and amplifications that increase CDK4/6 activity contribute to the progression of many cancers. However, when CDK4/...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | eLife |
|---|---|
| Κύριοι συγγραφείς: | , , , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
eLife Sciences Publications, Ltd
2020
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7213986/ https://ncbi.nlm.nih.gov/pubmed/32255427 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.44571 |
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