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Stress-mediated exit to quiescence restricted by increasing persistence in CDK4/6 activation
Mammalian cells typically start the cell-cycle entry program by activating cyclin-dependent protein kinase 4/6 (CDK4/6). CDK4/6 activity is clinically relevant as mutations, deletions, and amplifications that increase CDK4/6 activity contribute to the progression of many cancers. However, when CDK4/...
שמור ב:
| הוצא לאור ב: | eLife |
|---|---|
| Main Authors: | , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
eLife Sciences Publications, Ltd
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7213986/ https://ncbi.nlm.nih.gov/pubmed/32255427 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.44571 |
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