Administration of a 20-HETE synthesis inhibitor improves outcome in a rat model of pediatric traumatic brain injury

The arachidonic acid pathway metabolite 20-hydroxyeicosatetraenoic acid (20-HETE) contributes to ischemia/reperfusion brain injury. Inhibition of 20-HETE formation can protect the developing brain from global ischemia. Here, we examined whether treatment with the 20-HETE synthesis inhibitor N-hydrox...

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Bibliografiske detaljer
Udgivet i:Dev Neurosci
Main Authors: Shu, Shiyu, Zhang, Zhi, Spicer, Dawn, Kulikowicz, Ewa, Hu, Ke, Babapoor-Farrokhran, Savalan, Kannan, Sujatha, Koehler, Raymond C., Robertson, Courtney L.
Format: Artigo
Sprog:Inglês
Udgivet: 2019
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Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7044071/
https://ncbi.nlm.nih.gov/pubmed/31553983
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000500895
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