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Administration of a 20-HETE synthesis inhibitor improves outcome in a rat model of pediatric traumatic brain injury
The arachidonic acid pathway metabolite 20-hydroxyeicosatetraenoic acid (20-HETE) contributes to ischemia/reperfusion brain injury. Inhibition of 20-HETE formation can protect the developing brain from global ischemia. Here, we examined whether treatment with the 20-HETE synthesis inhibitor N-hydrox...
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| Publicado no: | Dev Neurosci |
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| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7044071/ https://ncbi.nlm.nih.gov/pubmed/31553983 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000500895 |
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