Loss of PI3 kinase association improves the sensitivity of secondary mutation of KIT to Imatinib

Lignende værker

• Complementary activity of tyrosine kinase inhibitors against secondary kit mutations in imatinib-resistant gastrointestinal stromal tumours
  af: Serrano, César, et al.
  Udgivet: (2019)
• Activation mutations of human c-KIT resistant to imatinib mesylate are sensitive to the tyrosine kinase inhibitor PKC412
  af: Growney, Joseph D., et al.
  Udgivet: (2005)
• Correction: Complementary activity of tyrosine kinase inhibitors against secondary kit mutations in imatinib-resistant gastrointestinal stromal tumours
  af: Serrano, César, et al.
  Udgivet: (2019)
• Secondary KIT mutations: the GIST of drug resistance and sensitivity
  af: Napolitano, Andrea, et al.
  Udgivet: (2019)
• Protein tyrosine phosphatase receptor type E (PTPRE) regulates the activation of wild-type KIT and KIT mutants differently
  af: Zhang, Shaoting, et al.
  Udgivet: (2021)