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Up‐regulation of HDACs, a harbinger of uraemic endothelial dysfunction, is prevented by defibrotide
Endothelial dysfunction is an earlier contributor to the development of atherosclerosis in chronic kidney disease (CKD), in which the role of epigenetic triggers cannot be ruled out. Endothelial protective strategies, such as defibrotide (DF), may be useful in this scenario. We evaluated changes ind...
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| Gepubliceerd in: | J Cell Mol Med |
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| Hoofdauteurs: | , , , , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
John Wiley and Sons Inc.
2019
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6991634/ https://ncbi.nlm.nih.gov/pubmed/31782253 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.14865 |
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