Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Ca(v)1.2 channel modulation under basal conditions

Neuronal nitric oxide synthase (nNOS) is considered a regulator of Ca(v)1.2 L-type Ca(2+) channels and downstream Ca(2+) cycling in the heart. The commonest view is that nitric oxide (NO), generated by nNOS activity in cardiomyocytes, reduces the currents through Ca(v)1.2 channels. This gives rise t...

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Опубликовано в: :Pflugers Arch
Главные авторы: Ebner, Janine, Cagalinec, Michal, Kubista, Helmut, Todt, Hannes, Szabo, Petra L., Kiss, Attila, Podesser, Bruno K., Cserne Szappanos, Henrietta, Hool, Livia C., Hilber, Karlheinz, Koenig, Xaver
Формат: Artigo
Язык:Inglês
Опубликовано: Springer Berlin Heidelberg 2019
Предметы:
Ion Channels, Receptors and Transporters
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC6960210/
https://ncbi.nlm.nih.gov/pubmed/31822999
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00424-019-02335-7
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