טוען...
Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Ca(v)1.2 channel modulation under basal conditions
Neuronal nitric oxide synthase (nNOS) is considered a regulator of Ca(v)1.2 L-type Ca(2+) channels and downstream Ca(2+) cycling in the heart. The commonest view is that nitric oxide (NO), generated by nNOS activity in cardiomyocytes, reduces the currents through Ca(v)1.2 channels. This gives rise t...
שמור ב:
| הוצא לאור ב: | Pflugers Arch |
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| Main Authors: | , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Springer Berlin Heidelberg
2019
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6960210/ https://ncbi.nlm.nih.gov/pubmed/31822999 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00424-019-02335-7 |
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