Perinatal protein restriction with postnatal catch-up growth leads to elevated p66Shc and mitochondrial dysfunction in the adult rat liver

Lignende værker

• In Utero Exposure to Δ9-Tetrahydrocannabinol Leads to Postnatal Catch-Up Growth and Dysmetabolism in the Adult Rat Liver
  af: Oke, Shelby L., et al.
  Udgivet: (2021)
• The Role of Cellular Stress in Intrauterine Growth Restriction and Postnatal Dysmetabolism
  af: Oke, Shelby L., et al.
  Udgivet: (2021)
• p66shc siRNA Nanoparticles Ameliorate Chondrocytic Mitochondrial Dysfunction in Osteoarthritis
  af: Shin, Hyo Jung, et al.
  Udgivet: (2020)
• Maternal Protein Restriction Elevates Cholesterol in Adult Rat Offspring Due to Repressive Changes in Histone Modifications at the Cholesterol 7α-Hydroxylase Promoter
  af: Sohi, Gurjeev, et al.
  Udgivet: (2011)
• Loss of PLA2G6 leads to elevated mitochondrial lipid peroxidation and mitochondrial dysfunction
  af: Kinghorn, Kerri J., et al.
  Udgivet: (2015)