Sam68 impedes the recovery of arterial injury by augmenting inflammatory response

OBJECTIVE: The role of Src-associated-in-mitosis-68-kDa (Sam68) in cardiovascular biology has not been studied. A recent report suggests that Sam68 promotes TNF-α–induced NF-κB activation in fibroblasts. Here we sought to dissect the molecular mechanism by which Sam68 regulates NF-κB signaling and i...

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Publicado en:J Mol Cell Cardiol
Autores principales: Han, Shuling, Xu, Shiyue, Zhou, Junlan, Qiao, Aijun, Boriboun, Chan, Ma, Wenxia, Li, Huadong, Biyashev, Dauren, Yang, Liu, Zhang, Eric, Liu, Qinghua, Jiang, Shayi, Zhao, Ting C, Krishnamurthy, Prasanna, Zhang, Chunxiang, Richard, Stéphane, Qiu, Hongyu, Zhang, Jianyi, Qin, Gangjian
Formato: Artigo
Lenguaje:Inglês
Publicado: 2019
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6889069/
https://ncbi.nlm.nih.gov/pubmed/31639388
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2019.10.003
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