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Sam68 impedes the recovery of arterial injury by augmenting inflammatory response
OBJECTIVE: The role of Src-associated-in-mitosis-68-kDa (Sam68) in cardiovascular biology has not been studied. A recent report suggests that Sam68 promotes TNF-α–induced NF-κB activation in fibroblasts. Here we sought to dissect the molecular mechanism by which Sam68 regulates NF-κB signaling and i...
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| Wydane w: | J Mol Cell Cardiol |
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| Główni autorzy: | , , , , , , , , , , , , , , , , , , |
| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
2019
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6889069/ https://ncbi.nlm.nih.gov/pubmed/31639388 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2019.10.003 |
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