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The anion transporter SLC26A9 localizes to tight junctions and is degraded by the proteasome when co-expressed with F508del–CFTR
Mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR) disrupt epithelial secretion and cause cystic fibrosis (CF). Available CFTR modulators provide only modest clinical benefits, so alternative therapeutic targets are being explored. The anion-conducting tran...
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| Publicat a: | J Biol Chem |
|---|---|
| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Biochemistry and Molecular Biology
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6885613/ https://ncbi.nlm.nih.gov/pubmed/31645438 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA119.010192 |
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