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Loss of SIM2s inhibits RAD51 binding and leads to unresolved replication stress
BACKGROUND: Mutations in genes associated with homologous recombination (HR) increase an individual’s risk of developing triple-negative breast cancer (TNBC). Although known for their role in repairing dsDNA breaks, HR repair elements also stabilize and restart stalled replication forks. Essential t...
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| 發表在: | Breast Cancer Res |
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| Main Authors: | , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
BioMed Central
2019
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6882179/ https://ncbi.nlm.nih.gov/pubmed/31775907 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13058-019-1207-z |
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