Juvenile Huntington’s Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein

Ítems similars

• The Proteasome Activators Blm10/PA200 Enhance the Proteasomal Degradation of N-Terminal Huntingtin
  per: Azzam Aladdin, et al.
  Publicat: (2020-11-01)
• The Proteasome Activators Blm10/PA200 Enhance the Proteasomal Degradation of N-Terminal Huntingtin
  per: Aladdin, Azzam, et al.
  Publicat: (2020)
• The proteasome activator PA200 regulates expression of genes involved in cell survival upon selective mitochondrial inhibition in neuroblastoma cells
  per: Douida, Abdennour, et al.
  Publicat: (2020)
• Cells Lacking PA200 Adapt to Mitochondrial Dysfunction by Enhancing Glycolysis via Distinct Opa1 Processing
  per: Douida, Abdennour, et al.
  Publicat: (2021)
• Postnatal and adult consequences of loss of huntingtin during development: Implications for Huntington’s disease
  per: Arteaga-Bracho, Eduardo E., et al.
  Publicat: (2016)