טוען...
Procyanidin B2 prevents lupus nephritis development in mice by inhibiting NLRP3 inflammasome activation
Lupus nephritis (LN) is a multifactorial event that contributes to the long-term mortality of systemic lupus erythematosus (SLE). Activation of NLRP3 inflammasome has been known to play a role in SLE pathogenesis. We evaluated the renal protection effects of procyanidin B2 (PCB2) and the involvement...
שמור ב:
| הוצא לאור ב: | Innate Immun |
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| Main Authors: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
SAGE Publications
2018
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6830913/ https://ncbi.nlm.nih.gov/pubmed/29874961 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1753425918780985 |
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