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Nitric Oxide Depresses GABA(A) Receptor Function via Coactivation of cGMP-Dependent Kinase and Phosphodiesterase

Nitric oxide (NO) is thought to play an essential role in neuronal processing, but the downstream mechanisms of its action remain unclear. We report here that NO analogs reduce GABA-gated currents in cultured retinal amacrine cells via two distinct, but convergent, cGMP-dependent pathways. Either ex...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:J Neurosci
मुख्य लेखकों: Wexler, Eric M., Stanton, Patric K., Nawy, Scott
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: Society for Neuroscience 1998
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6793112/
https://ncbi.nlm.nih.gov/pubmed/9502795
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.18-07-02342.1998
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