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Rapid Ca(2+) Entry through Ca(2+)-Permeable AMPA/Kainate Channels Triggers Marked Intracellular Ca(2+)Rises and Consequent Oxygen Radical Production
The widespread neuronal injury that results after brief activation of highly Ca(2+)-permeable NMDA channels may, in large part, reflect mitochondrial Ca(2+) overload and the consequent production of injurious oxygen radicals. In contrast, AMPA/kainate receptor activation generally causes slower toxi...
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| Gepubliceerd in: | J Neurosci |
|---|---|
| Hoofdauteurs: | , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Society for Neuroscience
1998
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6793031/ https://ncbi.nlm.nih.gov/pubmed/9742143 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.18-19-07727.1998 |
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