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Blockade of Ca(2+)-Permeable AMPA/Kainate Channels Decreases Oxygen–Glucose Deprivation-Induced Zn(2+)Accumulation and Neuronal Loss in Hippocampal Pyramidal Neurons
Synaptic release of Zn(2+) and its translocation into postsynaptic neurons probably contribute to neuronal injury after ischemia or epilepsy. Studies in cultured neurons have revealed that of the three major routes of divalent cation entry, NMDA channels, voltage-sensitive Ca(2+) channels (VSCCs), a...
Gorde:
| Argitaratua izan da: | J Neurosci |
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| Egile Nagusiak: | , , , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
Society for Neuroscience
2002
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6757569/ https://ncbi.nlm.nih.gov/pubmed/11850455 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-04-01273.2002 |
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