Blockade of Ca(2+)-Permeable AMPA/Kainate Channels Decreases Oxygen–Glucose Deprivation-Induced Zn(2+)Accumulation and Neuronal Loss in Hippocampal Pyramidal Neurons

Synaptic release of Zn(2+) and its translocation into postsynaptic neurons probably contribute to neuronal injury after ischemia or epilepsy. Studies in cultured neurons have revealed that of the three major routes of divalent cation entry, NMDA channels, voltage-sensitive Ca(2+) channels (VSCCs), a...

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Bibliographic Details
Published in:J Neurosci
Main Authors: Yin, Hong Z., Sensi, Stefano L., Ogoshi, Fumio, Weiss, John H.
Format: Artigo
Language:Inglês
Published: Society for Neuroscience 2002
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ARTICLE
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC6757569/
https://ncbi.nlm.nih.gov/pubmed/11850455
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.22-04-01273.2002
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