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N-Glycosylation of the Discoidin Domain Receptor Is Required for Axon Regeneration in Caenorhabditis elegans

Axon regeneration following neuronal injury is an important repair mechanism that is not well understood at present. In Caenorhabditis elegans, axon regeneration is regulated by DDR-2, a receptor tyrosine kinase (RTK) that contains a discoidin domain and modulates the Met-like SVH-2 RTK–JNK MAP kina...

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Библиографические подробности
Опубликовано в: :Genetics
Главные авторы: Shimizu, Tatsuhiro, Kato, Yuka, Sakai, Yoshiki, Hisamoto, Naoki, Matsumoto, Kunihiro
Формат: Artigo
Язык:Inglês
Опубликовано: Genetics Society of America 2019
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC6781908/
https://ncbi.nlm.nih.gov/pubmed/31371405
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1534/genetics.119.302492
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