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Is Alzheimer disease a failure of mobilizing immune defense? Lessons from cognitively fit oldest-old

Multifaceted evidence supports the hypothesis that inflammatory-immune mechanisms contribute to Alzheimer disease (AD) neuropathology and genetic association of several immune specific genes (TREM2, CR1, and CD33) suggests that maladaptive immune responses may be pivotal drivers of AD pathogenesis....

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Detalles Bibliográficos
Publicado en:Dialogues Clin Neurosci
Main Authors: Katsel, Pavel, Haroutunian, Vahram
Formato: Artigo
Idioma:Inglês
Publicado: Les Laboratoires Servier 2019
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC6780355/
https://ncbi.nlm.nih.gov/pubmed/31607776
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