Activation of NPRs and UCP1-independent pathway following CB1R antagonist treatment is associated with adipose tissue beiging in fat-fed male dogs

Samankaltaisia teoksia

• A Peripheral CB1R Antagonist Increases Lipolysis, Oxygen Consumption Rate, and Markers of Beiging in 3T3-L1 Adipocytes Similar to RIM, Suggesting that Central Effects Can Be Avoided
  Tekijä: Paszkiewicz, Rebecca L., et al.
  Julkaistu: (2020)
• Correction: Paszkiewicz et al. A Peripheral CB1R Antagonist Increases Lipolysis, Oxygen Consumption Rate, and Markers of Beiging in 3T3-L1 Adipocytes Similar to RIM, Suggesting That Central Effects Can Be Avoided. Int. J. Mol. Sci. 2020, 21, 6639
  Tekijä: Paszkiewicz, Rebecca L., et al.
  Julkaistu: (2021)
• CB1R antagonist increases hepatic insulin clearance in fat-fed dogs likely via upregulation of liver adiponectin receptors
  Tekijä: Kabir, Morvarid, et al.
  Julkaistu: (2015)
• Variability of Directly Measured First-Pass Hepatic Insulin Extraction and Its Association With Insulin Sensitivity and Plasma Insulin
  Tekijä: Asare-Bediako, Isaac, et al.
  Julkaistu: (2018)
• Renal Denervation Reverses Hepatic Insulin Resistance Induced by High-Fat Diet
  Tekijä: Iyer, Malini S., et al.
  Julkaistu: (2016)