Calpain Is a Major Cell Death Effector in Selective Striatal Degeneration Induced In Vivo by 3-Nitropropionate: Implications for Huntington's Disease

Striatal cell death in Huntington's Disease (HD) may involve mitochondrial defects, NMDA-mediated excitotoxicity, and activation of death effector proteases such as caspases and calpain. However, the precise contribution of mitochondrial defects in the activation of these proteases in HD is unk...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Bizat, Nicolas, Hermel, Jean-Michel, Boyer, Frédéric, Jacquard, Carine, Créminon, Christophe, Ouary, Stéphane, Escartin, Carole, Hantraye, Philippe, Krajewski, Stan, Brouillet, Emmanuel
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2003
Assuntos:
Cellular/Molecular
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6741191/
https://ncbi.nlm.nih.gov/pubmed/12832525
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.23-12-05020.2003
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