Chronic 3-Nitropropionic Acid Treatment in Baboons Replicates the Cognitive and Motor Deficits of Huntington’s Disease

We showed recently that chronic administration of the mitochondrial inhibitor 3-nitropropionic acid (3NP) in primates produces various dyskinetic movements and dystonic postures associated with selective striatal lesions displaying many similarities with the pathological features of Huntington’s dis...

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Pubblicato in:J Neurosci
Autori principali: Palfi, Stéphane, Ferrante, Robert J., Brouillet, Emmanuel, Beal, M. Flint, Dolan, Robert, Guyot, Marie Caroline, Peschanski, Marc, Hantraye, Philippe
Natura: Artigo
Lingua:Inglês
Pubblicazione: Society for Neuroscience 1996
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Articles
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6579050/
https://ncbi.nlm.nih.gov/pubmed/8622131
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.16-09-03019.1996
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