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Integrin α4β1 and TLR4 Cooperate to Induce Fibrotic Gene Expression in Response to Fibronectin’s EDA Domain
Alternative splicing of fibronectin increases expression of the EDA(+) isoform of fibronectin (EDA(+)Fn), a damage-associated molecular pattern (DAMP) molecule, which promotes fibro-inflammatory disease through the activation of toll-like receptors (TLR). Our studies indicate that the fibronectin ED...
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| Pubblicato in: | J Invest Dermatol |
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| Autori principali: | , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2017
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6736604/ https://ncbi.nlm.nih.gov/pubmed/28842322 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jid.2017.08.005 |
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