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Integrin α4β1 and TLR4 Cooperate to Induce Fibrotic Gene Expression in Response to Fibronectin’s EDA Domain

Alternative splicing of fibronectin increases expression of the EDA(+) isoform of fibronectin (EDA(+)Fn), a damage-associated molecular pattern (DAMP) molecule, which promotes fibro-inflammatory disease through the activation of toll-like receptors (TLR). Our studies indicate that the fibronectin ED...

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Bibliografske podrobnosti
izdano v:	J Invest Dermatol
Main Authors:	Kelsh-Lasher, Rhiannon M., Ambesi, Anthony, Bertram, Cedya, McKeown-Longo, Paula J.
Format:	Artigo
Jezik:	Inglês
Izdano:	2017
Teme:	Article
Online dostop:	https://ncbi.nlm.nih.gov/pmc/articles/PMC6736604/ https://ncbi.nlm.nih.gov/pubmed/28842322 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jid.2017.08.005
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Integrin α4β1 and TLR4 Cooperate to Induce Fibrotic Gene Expression in Response to Fibronectin’s EDA Domain

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