Propofol Restores the Function of “Hyperekplexic” Mutant Glycine Receptors in Xenopus Oocytes and Mice

Human hereditary hyperekplexia (“startle disease”) is a neurological disorder characterized by exaggerated, convulsive movements in response to unexpected stimuli. Molecular genetic studies have shown that this disease is often caused by amino acid substitutions at arginine 271 to glutamine or leuci...

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Bibliographische Detailangaben
Veröffentlicht in:J Neurosci
Hauptverfasser: O'Shea, Sean Michael, Becker, Lore, Weiher, Hans, Betz, Heinrich, Laube, Bodo
Format: Artigo
Sprache:Inglês
Veröffentlicht: Society for Neuroscience 2004
Schlagworte:
Brief Communications
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730415/
https://ncbi.nlm.nih.gov/pubmed/14999083
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4675-03.2004
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