Propofol Restores the Function of “Hyperekplexic” Mutant Glycine Receptors in Xenopus Oocytes and Mice

Human hereditary hyperekplexia (“startle disease”) is a neurological disorder characterized by exaggerated, convulsive movements in response to unexpected stimuli. Molecular genetic studies have shown that this disease is often caused by amino acid substitutions at arginine 271 to glutamine or leuci...

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Detaylı Bibliyografya
Yayımlandı:J Neurosci
Asıl Yazarlar: O'Shea, Sean Michael, Becker, Lore, Weiher, Hans, Betz, Heinrich, Laube, Bodo
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2004
Konular:
Brief Communications
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6730415/
https://ncbi.nlm.nih.gov/pubmed/14999083
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4675-03.2004
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