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Dysregulated IP(3) Signaling in Cortical Neurons of Knock-In Mice Expressing an Alzheimer's-Linked Mutation in Presenilin1 Results in Exaggerated Ca(2+) Signals and Altered Membrane Excitability
Disruptions in intracellular Ca(2+) signaling are proposed to underlie the pathophysiology of Alzheimer's disease (AD), and it has recently been shown that AD-linked mutations in the presenilin 1 gene (PS1) enhance inositol triphosphate (IP(3))-mediated Ca(2+) liberation in nonexcitable cells....
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| Udgivet i: | J Neurosci |
|---|---|
| Main Authors: | , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Society for Neuroscience
2004
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6729995/ https://ncbi.nlm.nih.gov/pubmed/14724250 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4386-03.2004 |
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