Mice Lacking Sodium Channel β1 Subunits Display Defects in Neuronal Excitability, Sodium Channel Expression, and Nodal Architecture

Sodium channel β1 subunits modulate α subunit gating and cell surface expression and participate in cell adhesive interactions in vitro. β1(-/-) mice appear ataxic and display spontaneous generalized seizures. In the optic nerve, the fastest components of the compound action potential are slowed and...

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Dades bibliogràfiques
Publicat a:J Neurosci
Autors principals: Chen, Chunling, Westenbroek, Ruth E., Xu, Xiaorong, Edwards, Chris A., Sorenson, Dorothy R., Chen, Yuan, McEwen, Dyke P., O'Malley, Heather A., Bharucha, Vandana, Meadows, Laurence S., Knudsen, Gabriel A., Vilaythong, Alex, Noebels, Jeffrey L., Saunders, Thomas L., Scheuer, Todd, Shrager, Peter, Catterall, William A., Isom, Lori L.
Format: Artigo
Idioma:Inglês
Publicat: Society for Neuroscience 2004
Matèries:
Cellular/Molecular
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6729427/
https://ncbi.nlm.nih.gov/pubmed/15102918
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4139-03.2004
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