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c-Jun NH(2)-Terminal Kinase-Interacting Protein-3 Facilitates Phosphorylation and Controls Localization of Amyloid-β Precursor Protein

Abnormal phosphorylation of amyloid-β precursor protein (APP) is a pathologic feature of Alzheimer's disease. To begin to understand the mechanism of APP phosphorylation, we studied this process in differentiating neurons under normal physiological conditions. We found that c-Jun NH(2)-terminal...

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Detalles Bibliográficos
Publicado en:J Neurosci
Autores principales: Muresan, Zoia, Muresan, Virgil
Formato: Artigo
Lenguaje:Inglês
Publicado: Society for Neuroscience 2005
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6724916/
https://ncbi.nlm.nih.gov/pubmed/15829626
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0152-05.2005
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