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Enhanced nuclear protein export in premature aging and rescue of the progeria phenotype by modulation of CRM1 activity
The study of Hutchinson–Gilford progeria syndrome (HGPS) has provided important clues to decipher mechanisms underlying aging. Progerin, a mutant lamin A, disrupts nuclear envelope structure/function, with further impairment of multiple processes that culminate in senescence. Here, we demonstrate th...
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| 出版年: | Aging Cell |
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| 主要な著者: | , , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2019
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6718587/ https://ncbi.nlm.nih.gov/pubmed/31305018 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.13002 |
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