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Methylglyoxal triggers human aortic endothelial cell dysfunction via modulation of the K(ATP)/MAPK pathway
Endothelial dysfunction is a key risk factor in diabetes-related multiorgan damage. Methylglyoxal (MGO), a highly reactive dicarbonyl generated primarily as a by-product of glycolysis, is increased in both type 1 and type 2 diabetic patients. MGO can rapidly bind with proteins, nucleic acids, and li...
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| Publicat a: | Am J Physiol Cell Physiol |
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| Autors principals: | , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6689756/ https://ncbi.nlm.nih.gov/pubmed/30995106 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00117.2018 |
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