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A novel animal model of emphysema induced by anti-elastin autoimmunity
Loss of immune tolerance to self-antigens can promote chronic inflammation and disrupt the normal function of multiple organs including the lungs. Degradation of elastin, a highly insoluble protein and a significant component of the lung structural matrix generates pro-inflammatory molecules. Elasti...
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| Publicado no: | J Immunol |
|---|---|
| Main Authors: | , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6688643/ https://ncbi.nlm.nih.gov/pubmed/31182478 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1900113 |
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