N-Terminally Deleted Forms of the Prion Protein Activate Both Bax-Dependent and Bax-Independent Neurotoxic Pathways

Lignende værker

• Bax deletion prevents neuronal loss but not neurological symptoms in a transgenic model of inherited prion disease
  af: Chiesa, Roberto, et al.
  Udgivet: (2005)
• Prion neurotoxicity
  af: Le, Nhat T. T., et al.
  Udgivet: (2019)
• An N-terminal Fragment of the Prion Protein Binds to Amyloid-β Oligomers and Inhibits Their Neurotoxicity in Vivo
  af: Fluharty, Brian R., et al.
  Udgivet: (2013)
• Both N-Terminal and C-Terminal Histidine Residues of the Prion Protein are Essential for Copper Coordination and Neuroprotective Self-Regulation.
  af: Schilling, Kevin M., et al.
  Udgivet: (2020)
• CYTOSOLIC PRION PROTEIN IS THE PREDOMINANT ANTI-BAX PRION PROTEIN FORM: EXCLUSION OF TRANSMEMBRANE AND SECRETED PRION PROTEIN FORMS IN THE ANTI-BAX FUNCTION
  af: Lin, David T. S., et al.
  Udgivet: (2008)