טוען...
Microglial Ca(2+)-Activated K(+) Channels Are Possible Molecular Targets for the Analgesic Effects of S-Ketamine on Neuropathic Pain
Ketamine is an important analgesia clinically used for both acute and chronic pain. The acute analgesic effects of ketamine are generally believed to be mediated by the inhibition of NMDA receptors in nociceptive neurons. However, the inhibition of neuronal NMDA receptors cannot fully account for it...
שמור ב:
| הוצא לאור ב: | J Neurosci |
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| Main Authors: | , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Society for Neuroscience
2011
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6623803/ https://ncbi.nlm.nih.gov/pubmed/22131399 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4152-11.2011 |
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