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Alteration of Synaptic Network Dynamics by the Intellectual Disability Protein PAK3
Several gene mutations linked to intellectual disability in humans code for synaptic molecules implicated in small GTPase signaling. This is the case of the Rac/Cdc42 effector p21-activated kinase 3 (PAK3). The mechanisms responsible for the intellectual defects and the consequences of the mutation...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Neurosci |
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| Κύριοι συγγραφείς: | , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Society for Neuroscience
2012
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6621069/ https://ncbi.nlm.nih.gov/pubmed/22238087 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3252-11.2012 |
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