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Recurrent noncoding mutations in skin cancers: UV damage susceptibility or repair inhibition as primary driver?

Somatic mutations arising in human skin cancers are heterogeneously distributed across the genome, meaning that certain genomic regions (e.g., heterochromatin or transcription factor binding sites) have much higher mutation densities than others. Regional variations in mutation rates are typically n...

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Bibliografiske detaljer
Udgivet i:Bioessays
Main Authors: Roberts, Steven A., Brown, Alexander J., Wyrick, John J.
Format: Artigo
Sprog:Inglês
Udgivet: 2019
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6571124/
https://ncbi.nlm.nih.gov/pubmed/30801747
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/bies.201800152
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