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mTORC1 and mTORC2 are differentially engaged in the development of laser-induced CNV
BACKGROUND: The mechanistic target of rapamycin (mTOR) pathway is a potential target to inhibit pathologic processes in choroidal neovascularization. However, the exact role of mTOR signaling in the development of CNV remains obscure. In this study, we assessed the role of mTORC1 and mTORC2 as well...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Cell Commun Signal |
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| Κύριοι συγγραφείς: | , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
BioMed Central
2019
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6570852/ https://ncbi.nlm.nih.gov/pubmed/31200728 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12964-019-0380-0 |
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