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Enhanced SIRT6 activity abrogates the neurotoxic phenotype of astrocytes expressing ALS-linked mutant SOD1

Sirtuins (SIRTs) are NAD(+)-dependent deacylases that play a key role in transcription, DNA repair, metabolism, and oxidative stress resistance. Increasing NAD(+) availability regulates endogenous SIRT activity, leading to increased resistance to oxidative stress and decreased mitochondrial reactive...

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Detalles Bibliográficos
Publicado en:FASEB J
Main Authors: Harlan, Benjamin A., Pehar, Mariana, Killoy, Kelby M., Vargas, Marcelo R.
Formato: Artigo
Idioma:Inglês
Publicado: Federation of American Societies for Experimental Biology 2019
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC6529338/
https://ncbi.nlm.nih.gov/pubmed/30841754
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201802752R
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