A code of mono-phosphorylation modulates the function of RB.

Hyper-phosphorylation of RB controls its interaction with E2F and inhibits its tumor suppressor properties. However, during G1 active RB can be mono-phosphorylated on any one of 14 CDK phosphorylation sites. Here we used quantitative proteomics to profile protein complexes formed by each mono-phosph...

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Detaylı Bibliyografya
Yayımlandı:Mol Cell
Asıl Yazarlar: Sanidas, Ioannis, Morris, Robert, Fella, Katerina A., Rumde, Purva H., Boukhali, Myriam, Tai, Eric C., Ting, David T., Lawrence, Michael S., Haas, Wilhelm, Dyson, Nicholas J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2019
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6424368/
https://ncbi.nlm.nih.gov/pubmed/30711375
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2019.01.004
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