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A code of mono-phosphorylation modulates the function of RB.
Hyper-phosphorylation of RB controls its interaction with E2F and inhibits its tumor suppressor properties. However, during G1 active RB can be mono-phosphorylated on any one of 14 CDK phosphorylation sites. Here we used quantitative proteomics to profile protein complexes formed by each mono-phosph...
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| Publicado en: | Mol Cell |
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| Main Authors: | , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2019
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6424368/ https://ncbi.nlm.nih.gov/pubmed/30711375 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2019.01.004 |
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