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Chronic treatment with the mitochondrial peptide humanin prevents age-related myocardial fibrosis in mice
Cardiac fibrosis is a biological process that increases with age and contributes to myocardial dysfunction. Humanin (HN) is an endogenous mitochondria-derived peptide that has cytoprotective effects and reduces oxidative stress. The present study aimed to test the hypothesis that chronic supplementa...
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| 出版年: | Am J Physiol Heart Circ Physiol |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6415743/ https://ncbi.nlm.nih.gov/pubmed/30004252 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00685.2017 |
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