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Apobec3A maintains HIV-1 latency through recruitment of epigenetic silencing machinery to the long terminal repeat
HIV-1 integrates into the genome of target cells and establishes latency indefinitely. Understanding the molecular mechanism of HIV-1 latency maintenance is needed for therapeutic strategies to combat existing infection. In this study, we found an unexpected role for Apobec3A (apolipoprotein B MRNA...
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| Gepubliceerd in: | Proc Natl Acad Sci U S A |
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| Hoofdauteurs: | , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
National Academy of Sciences
2019
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6369738/ https://ncbi.nlm.nih.gov/pubmed/30670656 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1819386116 |
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