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HSP90 inhibitor 17-DMAG effectively alleviated the progress of thoracic aortic dissection by suppressing smooth muscle cell phenotypic switch
Thoracic aortic dissection (TAD) is a highly lethal vascular disease characterized by medial degeneration. Heat shock protein 90 (HSP90) had been proved as a potential target for a variety of diseases. The aim of this study was to identify the effect of HSP90 inhibitor on TAD progress, and to explor...
Zapisane w:
Wydane w: | Am J Transl Res |
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Główni autorzy: | , , , , , , , , |
Format: | Artigo |
Język: | Inglês |
Wydane: |
e-Century Publishing Corporation
2019
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Hasła przedmiotowe: | |
Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6357309/ https://ncbi.nlm.nih.gov/pubmed/30788006 |
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