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Repression of Transcription at DNA Breaks Requires Cohesin throughout Interphase and Prevents Genome Instability
Cohesin subunits are frequently mutated in cancer, but how they function as tumor suppressors is unknown. Cohesin mediates sister chromatid cohesion, but this is not always perturbed in cancer cells. Here, we identify a previously unknown role for cohesin. We find that cohesin is required to repress...
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| 出版年: | Mol Cell |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Cell Press
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6344341/ https://ncbi.nlm.nih.gov/pubmed/30554942 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2018.11.001 |
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