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Activation of c-Met in cancer cells mediates growth-promoting signals against oxidative stress through Nrf2-HO-1
Any imbalance between reactive oxygen species (ROS) generation and the anti-oxidant capacity lead to cellular oxidative stress. Many chemotherapeutic agents mediate their cytotoxic functions through the generation of ROS. c-Met, a receptor tyrosine kinase, is over-expressed in renal cancer and plays...
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| Publicado no: | Oncogenesis |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6333845/ https://ncbi.nlm.nih.gov/pubmed/30647407 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41389-018-0116-9 |
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