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Mitochondrial Dysfunction in HIV-1 Transgenic Mouse Cardiac Myocytes
The pathophysiology of human immunodeficiency virus (HIV)-associated cardiomyopathy remains uncertain. We used HIV-1 transgenic (Tg26) mice to explore mechanisms by which HIV-related proteins impacted on myocyte function. Compared to adult ventricular myocytes isolated from non-transgenic (WT) litte...
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| I publikationen: | J Cell Physiol |
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| Huvudupphovsmän: | , , , , , , , , , , , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2018
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6318060/ https://ncbi.nlm.nih.gov/pubmed/30256393 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jcp.27232 |
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